NBC to replace “Today Show” producer, source says
















(Reuters) – NBC is expected to name Alexandra Wallace, a senior vice president of the network’s news division, as the executive in charge of “The Today Show,” the latest reshuffling of the show’s personnel after it slipped to second in ratings this year behind “Good Morning America.”


Wallace, who would be the first woman in charge of the long-running NBC show that pioneered early morning TV in the United States, will be named along with a producer to replace Jim Bell, according to a person familiar with the decision.













Bell, who has headed the show since 2005, was blamed this year for the controversial firing of Ann Curry as anchor alongside Matt Lauer.


Curry was replaced by Savannah Guthrie in June.


“Good Morning America” or GMA, produced by Walt Disney‘s ABC unit, closed the gap with “Today.”


“Today,” the top-rated morning show for 16 consecutive years, started the current TV season number two. In late October, NBC drew 7,000 more viewers than GMA among 25 to 54 year-old viewers, the age group advertisers most want to reach, its first lead since September 10. GMA still led among overall viewers.


The first two hours of “The Today Show,” from 7 a.m. to 9 a.m., collected $ 485 million in ad revenues in 2011, up 6.6 percent from 2010, according to Kantar Media, which provides data to advertisers. GMA took in $ 299 million last year.


It is unclear when the changes at “The Today Show” will take effect, according to The New York Times, which first reported the shakeup.


Bell this summer produced NBC’s Summer Olympics coverage and is expected to become the full-time executive producer of the network’s ongoing Olympic coverage.


NBC, a unit of Comcast Corp., is also in the midst of layoffs at its entertainment unit, shedding 500 positions primarily at its cable channels. Jay Leno’s late night TV show cut about two dozen of its crew members about two months ago.


(Reporting By Ronald Grover)


TV News Headlines – Yahoo! News



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For Alzheimer’s, Detection Advances Outpace Treatment Options


Joshua Lott for The New York Times


Awilda Jimenez got a scan for Alzheimer’s after she started forgetting things. It was positive.







When Awilda Jimenez started forgetting things last year, her husband, Edwin, felt a shiver of dread. Her mother had developed Alzheimer’s in her 50s. Could his wife, 61, have it, too?




He learned there was a new brain scan to diagnose the disease and nervously agreed to get her one, secretly hoping it would lay his fears to rest. In June, his wife became what her doctor says is the first private patient in Arizona to have the test.


“The scan was floridly positive,” said her doctor, Adam S. Fleisher, director of brain imaging at the Banner Alzheimer’s Institute in Phoenix.


The Jimenezes have struggled ever since to deal with this devastating news. They are confronting a problem of the new era of Alzheimer’s research: The ability to detect the disease has leapt far ahead of treatments. There are none that can stop or even significantly slow the inexorable progression to dementia and death.


Families like the Jimenezes, with no good options, can only ask: Should they live their lives differently, get their affairs in order, join a clinical trial of an experimental drug?


“I was hoping the scan would be negative,” Mr. Jimenez said. “When I found out it was positive, my heart sank.”


The new brain scan technology, which went on the market in June, is spreading fast. There are already more than 300 hospitals and imaging centers, located in most major metropolitan areas, that are ready to perform the scans, according to Eli Lilly, which sells the tracer used to mark plaque for the scan.


The scans show plaques in the brain — barnaclelike clumps of protein, beta amyloid — that, together with dementia, are the defining feature of Alzheimer’s disease. Those who have dementia but do not have excessive plaques do not have Alzheimer’s. It is no longer necessary to wait until the person dies and has an autopsy to learn if the brain was studded with plaques.


Many insurers, including Medicare, will not yet pay for the new scans, which cost several thousand dollars. And getting one comes with serious risks. While federal law prevents insurers and employers from discriminating based on genetic tests, it does not apply to scans. People with brain plaques can be denied long-term care insurance.


The Food and Drug Administration, worried about interpretations of the scans, has required something new: Doctors must take a test showing they can read them accurately before they begin doing them. So far, 700 doctors have qualified, according to Eli Lilly. Other kinds of diagnostic scans have no such requirement.


In another unusual feature, the F.D.A. requires that radiologists not be told anything about the patient. They are generally trained to incorporate clinical information into their interpretation of other types of scans, said Dr. R. Dwaine Rieves, director of the drug agency’s Division of Medical Imaging Products.


But in this case, clinical information may lead radiologists to inadvertently shade their reports to coincide with what doctors suspect is the underlying disease. With Alzheimer’s, Dr. Rieves said, “clinical impressions have been misleading.”


“This is a big change in the world of image interpretation,” he said.


Like some other Alzheimer’s experts, Dr. Fleisher used the amyloid scan for several years as part of a research study that led to its F.D.A. approval. Subjects were not told what the scans showed. Now, with the scan on the market, the rules have changed.


Dr. Fleisher’s first patient was Mrs. Jimenez. Her husband, the family breadwinner, had lost his job as a computer consultant when the couple moved from New York to Arizona to take care of Mrs. Jimenez’s mother. Paying several thousand dollars for a scan was out of the question. But Dr. Fleisher found a radiologist, Dr. Mantej Singh Sra of Sun Radiology, who was so eager to get into the business that he agreed to do Mrs. Jimenez’s scan free. His plan was to be the first in Arizona to do a scan, and advertise it.


After Dr. Sra did the scan, the Jimenezes returned to Dr. Fleisher to learn the result.


Dr. Fleisher, sad to see so much plaque in Mrs. Jimenez’s brain, referred her to a psychiatrist to help with anxiety and suggested she enter clinical trials of experimental drugs.


But Mr. Jimenez did not like that idea. He worried about unexpected side effects.


“Tempting as it is, where do you draw the line?” he asks. “At what point do you take a risk with a loved one?”


At Mount Sinai Medical Center in New York, Dr. Samuel E. Gandy found that his patients — mostly affluent — were unfazed by the medical center’s $3,750 price for the scan. He has been ordering at least one a week for people with symptoms ambiguous enough to suggest the possibility of brain plaques.


Most of his patients want their names kept confidential, fearing an inability to get long-term care insurance, or just wanting privacy.


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L.A. County sheriff's deputy held in fatal off-duty shooting









A veteran Los Angeles County sheriff's deputy was arrested Wednesday for allegedly shooting and killing a man in Sylmar while off-duty in June, authorities said.

The deputy, Francisco Gamez, 41, has been with the department for 17 years and was last working as a station detective in West Hollywood.

Law enforcement sources told The Times that the deputy's son got into a dispute with another person. The son, they said, called his father to the scene. The deputy allegedly drove up soon after and exchanged words before opening fire from inside his car, striking one man, the sources said.





He then allegedly drove a short distance before shooting at a second person, added the sources, who asked for anonymity because the investigation is ongoing.

That person was not injured, according to authorities.

The other victim, Armando Casillas, 38, was taken to a hospital, where he was pronounced dead from a gunshot wound to the chest just before midnight on June 17.


FOR THE RECORD:
An earlier version of this article misspelled the victim's first name as Armondo.

Neighbors said Gamez and Casillas lived a block apart.

In August, a person who identified himself as the victim's brother commented on the website of the Los Angeles Times, saying he suspected a deputy was responsible.

"We think he is a L.A. COUNTY SHERIFF," the comment stated. "The reason we think he is a Sheriff is that he shouted to my Brother "L.A. COUNTY SHERIFF WHERE YOU FROM" as if the sheriff was in a gang."

The person who wrote the comment could not be reached Wednesday evening.

At the time of the killing, authorities said the victim got into an argument with an unknown person. At some point, the other person left the area only to return and shoot Casillas in a drive-by, authorities said then. Now they are saying that the shooter was not the same person who initially got into the argument.

LAPD officers arrested Gamez on suspicion of murder, attempted murder and use of a firearm in the commission of a felony. He was booked into the LAPD's 77th Street station Wednesday in lieu of $4-million bail, officials said. He has not been charged.

Casillas' sister said that the family was thankful for the arrest, but that they were not prepared to discuss the events that led to the fatal shooting.

In a statement, Sheriff Lee Baca called the incident "deeply disturbing."

His spokesman Steve Whitmore said the department placed Gamez on leave July 3 after learning from the LAPD about the investigation.

"He's been stripped of all law enforcement power," Whitmore said. "It casts a pall over the scores and scores of deputy sheriffs that every day do their job."

robert.faturechi@latimes.com

richard.winton@latimes.com

Times staff writer Andrew Blankstein contributed to this report.





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Kill the Password: Why a String of Characters Can't Protect Us Anymore



You have a secret that can ruin your life.


It’s not a well-kept secret, either. Just a simple string of characters—maybe six of them if you’re careless, 16 if you’re cautious—that can reveal everything about you.


Your email. Your bank account. Your address and credit card number. Photos of your kids or, worse, of yourself, naked. The precise location where you’re sitting right now as you read these words. Since the dawn of the information age, we’ve bought into the idea that a password, so long as it’s elaborate enough, is an adequate means of protecting all this precious data. But in 2012 that’s a fallacy, a fantasy, an outdated sales pitch. And anyone who still mouths it is a sucker—or someone who takes you for one.


No matter how complex, no matter how unique, your passwords can no longer protect you.


Look around. Leaks and dumps—hackers breaking into computer systems and releasing lists of usernames and passwords on the open web—are now regular occurrences. The way we daisy-chain accounts, with our email address doubling as a universal username, creates a single point of failure that can be exploited with devastating results. Thanks to an explosion of personal information being stored in the cloud, tricking customer service agents into resetting passwords has never been easier. All a hacker has to do is use personal information that’s publicly available on one service to gain entry into another.


This summer, hackers destroyed my entire digital life in the span of an hour. My Apple, Twitter, and Gmail passwords were all robust—seven, 10, and 19 characters, respectively, all alphanumeric, some with symbols thrown in as well—but the three accounts were linked, so once the hackers had conned their way into one, they had them all. They really just wanted my Twitter handle: @mat. As a three-letter username, it’s considered prestigious. And to delay me from getting it back, they used my Apple account to wipe every one of my devices, my iPhone and iPad and MacBook, deleting all my messages and documents and every picture I’d ever taken of my 18-month-old daughter.


The age of the password is over. We just haven’t realized it yet.


Since that awful day, I’ve devoted myself to researching the world of online security. And what I have found is utterly terrifying. Our digital lives are simply too easy to crack. Imagine that I want to get into your email. Let’s say you’re on AOL. All I need to do is go to the website and supply your name plus maybe the city you were born in, info that’s easy to find in the age of Google. With that, AOL gives me a password reset, and I can log in as you.


First thing I do? Search for the word “bank” to figure out where you do your online banking. I go there and click on the Forgot Password? link. I get the password reset and log in to your account, which I control. Now I own your checking account as well as your email.


This summer I learned how to get into, well, everything. With two minutes and $4 to spend at a sketchy foreign website, I could report back with your credit card, phone, and Social Security numbers and your home address. Allow me five minutes more and I could be inside your accounts for, say, Amazon, Best Buy, Hulu, Microsoft, and Netflix. With yet 10 more, I could take over your AT&T, Comcast, and Verizon. Give me 20—total—and I own your PayPal. Some of those security holes are plugged now. But not all, and new ones are discovered every day.


The common weakness in these hacks is the password. It’s an artifact from a time when our computers were not hyper-connected. Today, nothing you do, no precaution you take, no long or random string of characters can stop a truly dedicated and devious individual from cracking your account. The age of the password has come to an end; we just haven’t realized it yet.


Passwords are as old as civilization. And for as long as they’ve existed, people have been breaking them.


In 413 BC, at the height of the Peloponnesian War, the Athenian general Demosthenes landed in Sicily with 5,000 soldiers to assist in the attack on Syracusae. Things were looking good for the Greeks. Syracusae, a key ally of Sparta, seemed sure to fall.


But during a chaotic nighttime battle at Epipole, Demosthenes’ forces were scattered, and while attempting to regroup they began calling out their watchword, a prearranged term that would identify soldiers as friendly. The Syracusans picked up on the code and passed it quietly through their ranks. At times when the Greeks looked too formidable, the watchword allowed their opponents to pose as allies. Employing this ruse, the undermatched Syracusans decimated the invaders, and when the sun rose, their cavalry mopped up the rest. It was a turning point in the war.


The first computers to use passwords were likely those in MIT’s Compatible Time-Sharing System, developed in 1961. To limit the time any one user could spend on the system, CTSS used a login to ration access. It only took until 1962 when a PhD student named Allan Scherr, wanting more than his four-hour allotment, defeated the login with a simple hack: He located the file containing the passwords and printed out all of them. After that, he got as much time as he wanted.


During the formative years of the web, as we all went online, passwords worked pretty well. This was due largely to how little data they actually needed to protect. Our passwords were limited to a handful of applications: an ISP for email and maybe an ecommerce site or two. Because almost no personal information was in the cloud—the cloud was barely a wisp at that point—there was little payoff for breaking into an individual’s accounts; the serious hackers were still going after big corporate systems.


So we were lulled into complacency. Email addresses morphed into a sort of universal login, serving as our username just about everywhere. This practice persisted even as the number of accounts—the number of failure points—grew exponentially. Web-based email was the gateway to a new slate of cloud apps. We began banking in the cloud, tracking our finances in the cloud, and doing our taxes in the cloud. We stashed our photos, our documents, our data in the cloud.


Eventually, as the number of epic hacks increased, we started to lean on a curious psychological crutch: the notion of the “strong” password. It’s the compromise that growing web companies came up with to keep people signing up and entrusting data to their sites. It’s the Band-Aid that’s now being washed away in a river of blood.


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Michael Jackson’s assistant files class-action lawsuit against “This Is It” tour promoter
















LOS ANGELES (TheWrap.com) – Michael Jackson has been dead for more than three years now – but apparently he lives on in the halls of America’s legal system.


Jackson’s former assistant, Michael Amir Williams, filed a class-action lawsuit against concert promoters AEG Live in Los Angeles Superior Court on Friday, claiming he and others hired to attend to the “Beat It” singer during his would-be “This Is It” tour at London’s O2 Arena were deprived of at least $ 7.5 million dollars in pay.













According to the suit, AEG was responsible for the financial loss because it hired Dr. Conrad Murray — who was found guilty of causing the singer’s death – to care for Jackson.


The suit claims that Jackson “bargained for the addition of Class to help Michael Jackson give the ‘first class performance’ as required by Contract. The express terms of the Contract allowed for class to be paid by AEG up to $ 7.5 million and any amount over $ 7.5 million to be paid for by Michael Jackson.”


Unfortunately, AEG also hired Murray, who administered a fatal dose of Propofol to Jackson in June 2009, before the concerts could take place. (Murray was convicted of involuntary manslaughter for Jackson’s death in November 2011.)


AEG’s lawyer, Marvin Putnam of O’Melveny & Myers, calls the lawsuit “frivolous” and “truly unfortunate.”


“This lawsuit is clearly frivolous; it is literally barred by at least four different legal doctrines,” Putnam said in a statement provided to TheWrap. “The easiest is that Mr. Williams was a personal employee of Michael Jackson’s, and was never a beneficiary of Mr. Jackson’s contract with AEG Live. As such he has no legal standing to sue on that contract. Nor can he legally bring a claim for Mr. Jackson’s wrongful death. The idea that Mr. Williams purports to sue on behalf of the many persons who did enter into relationships with AEG Live and Jackson in connection with the This Is It Tour, and with whom AEG Live parted ways with the utmost friendship and respect, is disgraceful. It is truly unfortunate that so many see Mr. Jackson’s demise as an opportunity to grab as much for themselves as possible. This is just the latest wrongful death lawsuit with someone hoping to profit from Michael Jackson’s tragic death in the same way they profited from his life.”


Williams’ suit alleges breach of express terms of contract; breach of implied terms of contract; and breach of implied covenant of good faith and fair dealing. The complaint seeks unspecified damages, plus court costs and attorneys’ fees.


(Pamela Chelin contributed to this report)


Music News Headlines – Yahoo! News



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Alzheimer’s Tied to Mutation Harming Immune Response





Alzheimer’s researchers and drug companies have for years concentrated on one hallmark of Alzheimer’s disease: the production of toxic shards of a protein that accumulate in plaques on the brain.




But now, in a surprising coincidence, two groups of researchers working from entirely different starting points have converged on a mutated gene involved in another aspect of Alzheimer’s disease: the immune system’s role in protecting against the disease. The mutation is suspected of interfering with the brain’s ability to prevent the buildup of plaque.


The discovery, researchers say, provides clues to how and why the disease progresses. The gene, known as TREM2, is only the second found to increase Alzheimer’s risk substantially in older people.


“It points very specifically to a potential metabolic pathway that you could intervene in to change the course of Alzheimer’s disease,” said William Thies, chief medical and scientific officer of the Alzheimer’s Association.


Much work remains to be done before scientists understand precisely how the newly discovered gene mutation leads to Alzheimer’s, but already there are some indications from studies in mice. When the gene is not mutated, white blood cells in the brain spring into action, gobbling up and eliminating the plaque-forming toxic protein, beta amyloid. As a result, Alzheimer’s can be staved off or averted.


But when the gene is mutated, the brain’s white blood cells are hobbled, making them less effective in their attack on beta amyloid.


People with the mutated gene have a threefold to fivefold increase in the likelihood of developing Alzheimer’s disease in old age.


The intact gene, says John Hardy of University College London, “is a safety net.” And those with the mutation, he adds, “are living life without a safety net.” Dr. Hardy is lead author of one of the papers.


The discovery also suggests that a new type of drug could be developed to enhance the gene’s activity, perhaps allowing the brain’s white blood cells to do their work.


“The field is in desperate need of new therapeutic agents,” said Alison Goate, an Alzheimer’s researcher at Washington University in St. Louis who contributed data to Dr. Hardy’s study. “This will give us an alternative approach.”


The fact that two research groups converged on the same gene gives experts confidence in the findings. Both studies were published online Wednesday in The New England Journal of Medicine. “Together they make a good case that this really is an Alzheimer’s gene,” said Gerard Schellenberg, an Alzheimer’s researcher at the University of Pennsylvania who was not involved with the work.


The other gene found to raise the odds that a person will get Alzheimer’s, ApoE4, is much more common and confers about the same risk as the mutated version of TREM2. But it is still not clear why ApoE4, discovered in 1993, makes Alzheimer’s more likely.


Because the mutations in the newly discovered gene are rare, occurring in no more than 2 percent of Alzheimer’s patients, it makes no sense to start screening people for them, Dr. Thies said. Instead, the discovery provides new clues to the workings of Alzheimer’s disease.


To find the gene, a research group led by Dr. Kari Stefansson of deCODE Genetics of Iceland started with a simple question.


“We asked, ‘Can we find anything in the genome that separates those who are admitted to nursing homes before the age of 75 and those who are still living at home at 85?’ ” he said.


Scientists searched the genomes of 2,261 Icelanders and zeroed in on TREM2. Mutations in that gene were more common among people with Alzheimer’s, as well as those who did not have an Alzheimer’s diagnosis but who had memory problems and might be on their way to developing Alzheimer’s.


The researchers confirmed their results by looking for the gene in people with and without Alzheimer’s in populations studied at Emory University, as well as in Norway, the Netherlands and Germany.


The TREM2 connection surprised Dr. Stefansson. Although researchers have long noticed that the brain is inflamed in Alzheimer’s patients, he had dismissed inflammation as a major factor in the disease.


“I was of the opinion that the immune system would play a fairly small role, if any, in Alzheimer’s disease,” Dr. Stefansson said. “This discovery cured me of that bias.”


Meanwhile, Dr. Hardy and Rita Guerreiro at University College London, along with Andrew Singleton at the National Institute on Aging, were intrigued by a strange, rare disease. Only a few patients had been identified, but their symptoms were striking. They had crumbling bones and an unusual dementia, sclerosing leukoencephalopathy.


“It’s a weird disease,” Dr. Hardy said.


He saw one patient in her 30s whose brain disease manifested in sexually inappropriate behavior. Also, her bones kept breaking. The disease was caused by mutations that disabled both the copy of TREM2 that she had inherited from her mother and the one from her father.


Eventually the researchers searched for people who had a mutation in just one copy of TREM2. To their surprise, it turned out that these people were likely to have Alzheimer’s disease.


They then asked researchers around the world who had genetic data from people with and without Alzheimer’s to look for TREM2 mutations.


“Sure enough, they had good evidence,” Dr. Hardy said. The mutations occurred in one-half of 1 percent of the general population but in 1 to 2 percent of patients with Alzheimer’s disease.


“That is a big effect,” Dr. Hardy said.


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If soldier killed Afghan civilians in rampage, did he act alone?









JOINT BASE LEWIS MCCHORD, Wash. — The case against U.S. Army Staff Sgt. Robert Bales appeared at first to be horribly simple: Days after a bomb blew off the leg of a friend in southern Afghanistan, the 39-year-old combat veteran picked up his rifle, a pistol and a grenade launcher, walked to two villages and allegedly conducted a one-man campaign of vengeance, killing 16 civilians and wounding six more.

But by the time a weeklong hearing wound up Tuesday in a military courtroom, one of the Army's highest-profile war crimes from Afghanistan raised almost as many questions as it answered.

If Bales acted alone, why were shots heard coming from one of the villages 20 minutes after he was spotted returning to the base for the first time?





Why was a witness who claimed to have seen two U.S. soldiers killing her husband while helicopters flew overhead not brought in to testify?

Why were the crime scenes so very different — at one house, a surgical execution of the head of the family, while at the next house, 11 men, women and children were shot and possibly stabbed, their bodies piled in a heap and burned?

The Article 32 hearing, held to determine whether there was evidence to hold the serviceman for a court-martial, drew a portrait not just of a rogue soldier, defense lawyers said. It revealed a U.S. combat outpost at which soldiers spent their evenings drinking alcohol, snorting Valium and taking steroids, all three of which Bales apparently had done before asking a friend to "take care of my kids" and setting out into the darkness outside the base perimeter, laughing.

"We have a dysfunctional, drinking and drugging … team," civilian defense lawyer Emma Scanlan said in her closing argument.

"We can't isolate Sgt. Bales within a bubble," Scanlan said, noting that he was under the supervision of Special Forces officers. "They are the command. They are in charge. And they are terrible at it."

Army prosecutors claim Bales acted alone and with chilling rationality: walking to the village of Alkozai, where he is accused of killing four people and wounding six, coming back to the base and telling a friend what he had done, then venturing out again to the village of Najiban, where he is accused of killing 12.

When Bales returned to Camp Belambay the second time, he admitted to friends he had done some "sick" things, and told them they would thank him when fighting season got underway again during warmer weather later in the spring, said prosecutor Maj. Robert Stelle.

"Terrible, terrible things happened. That is clear. The second thing that is clear is that Staff Sgt. Bales did it," Stelle said.

He urged investigating officer Col. Lee Deneke to recommend a full court-martial and that it be tried as a capital case, with the possibility of the death penalty. Bales committed "the worst, most despicable crime a human being can commit: murdering children in their own homes," Stelle said.

If Deneke recommends a court-martial, a lengthy process of mental health evaluations and further forensic reports will follow. Defense lawyers have pledged there will be a full exploration of the medical care Bales received at the Madigan Army Medical Center for an earlier traumatic brain injury.

The hospital south of Seattle was the subject of an investigation this year for tossing aside diagnoses of post-traumatic stress disorder, making it harder for those soldiers to receive follow-up medical care.

In the year before the shootings, Scanlan said, Bales had received an exemplary evaluation, in which he was praised for "a strong moral compass, never wavering from what was right."

What went wrong?

Scanlan suggested part of the answer might lie in the steroids and sleep aids Bales was taking, along with the Jack Daniels he drank with two friends on the evening before the killings as they watched "Man on Fire," a movie about a former CIA operative who executes a violent campaign of revenge.

"They drank a ton, and they were all drunk," Scanlan said, noting that testimony showed that one of the soldiers who'd been drinking with Bales that night, Cpl. David Godwin, was stumbling, slurring his words and smelled like alcohol four hours later.

The prosecution said Bales had plenty of time to reach both villages and return at the end of the night with his clothing and weapons covered in blood. Before he left, prosecutors said, he had discussed his frustration with the Army's lack of response to the bomb that blew off his friend's leg; after his arrest, he reminded one soldier of an Afghan machine gunner the unit had faced earlier.

"That's not going to happen again," Bales said, according to Staff Sgt. Ross O'Rourke.

Prosecutors also established that DNA from at least one of the women killed at the home of Haji Mohammed Wazir in Najiban was found on Bales' clothing.

Yet defense lawyers continue to raise questions about whether Bales was the only one responsible — whether one person could have killed so many people in so many locations in one night.

A lot of the evidence suggests otherwise, Scanlan said, pointing to an agent from the Army's Criminal Investigations Command who said that Masuma Dawood, whose husband was shot, told her that two soldiers had killed her husband.

Army officials said Dawood did not testify because of "cultural differences," and the reluctance of Afghan families to allow a woman to testify in an American courtroom, even by remote video from Afghanistan. But sources in Afghanistan have told the Los Angeles Times that Dawood was, in fact, willing to testify.

Scanlan said the timeline laid out by prosecutors also raises questions, beginning with the Afghan guard who testified that he checked his watch, and was certain that the U.S. soldier he saw — returning from the initial killings in Alkozai, prosecutors allege — had returned to Camp Belambay at 1:30 a.m.

The shots heard from the direction of Alkozai didn't stop till 1:50 a.m., the defense attorney said.

"I don't know what that means," Scanlan said. "But one thing it means is, if you believe what the government is telling you, that Sgt. Bales is the one who came back through that wire at 1:30, then somebody else was firing for another 20 minutes."

kim.murphy@latimes.com





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Still on the Lam, John McAfee Says He's Now In Disguise



It doesn’t sound like much of a disguise, but John McAfee is doing his best to change his appearance as he continues to evade the police in Belize.


In a case that seems to get more bizarre by the day, the 67-year-old has continued to call me with semi-hourly updates. The latest disclosure: He claims to have dyed his hair, eyebrows, beard, and mustache jet black.


“I have modified my appearance in a radical fashion,” McAfee said, “I’ll probably look like a murderer, unfortunately.”


The American antivirus pioneer is wanted for questioning in connection with the murder of Gregory Faull, 52, an American expatriate and neighbor of McAfee’s. They both have beachside properties on the island of Ambergris Caye, about 20 miles off the Belize coast. Faull was found dead, face up in a pool of blood, in his villa Sunday morning, shot once in the back of the head. Faull had complained about the barking of McAfee’s dogs — McAfee kept 11 at his beachside compound — and four of those dogs were poisoned Friday night.


When police arrived at McAfee’s property Sunday afternoon to question him, McAfee hid, he says, burying himself in sand and covering his head with a cardboard box. He says he spent 18 hours hiding on his property before slipping away.


He has been running ever since, he says, riding in boats, huddling on the floorboards of taxis, sleeping in a bed that he said was infested with lice.


Since going into hiding, McAfee has repeatedly denied that he had anything to do with Faull’s death. He says that he does not want to give himself up to authorities because he is afraid they will torture or kill him.


He’s convinced that, while he’s away from his compound, the police will plant incriminating evidence unrelated to Faull’s murder. “The police have been to my house seven times,” he said. “I expect them to uncover a cache of fully automatic weapons, four tons of cocaine. Maybe a Soviet submarine.”


The authorities, meanwhile appear to be trying to put pressure on McAfee by arresting friends and associates, according to two sources (who are not McAfee). Since Sunday, police have detained one of McAfee’s bodyguards, William Mulligan; his groundskeeper, Cassian Chavaria; and a local taxi driver, Cesar Trapp.


McAfee said he is outraged by the detentions: “This is exactly what happened to Soviet dissidents when Stalin took power. If they could not catch the man himself, they rounded up all of his friends.”


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Accuser recants sex claims against Elmo puppeteer: report
















LOS ANGELES (Reuters) – The man who claimed he had underage sex with the puppeteer behind “Sesame Street” character Elmo recanted his claims on Tuesday, U.S. media reported.


The unnamed man, now 23, had claimed that Elmo puppeteer Kevin Clash had a sexual relationship with him when the accuser was 16 years old, potentially engulfing one of the biggest childhood brands in an underage sex scandal.













“He wants it to be known that his sexual relationship with Mr. Clash was an adult consensual relationship,” the law firm Andreozzi and Associates, who represent the man, told U.S. media outlets in a statement.


Clash, 52, who had denied the allegations, said in a statement obtained by Reuters on Tuesday: “I am relieved that this painful allegation has been put to rest. I will not discuss it further.”


New York-based Sesame Workshop said on Monday that its own inquiry had concluded that the claims of underage sexual conduct against Clash were unsubstantiated.


“We are pleased that this matter has been brought to a close, and we are happy that Kevin can move on from this unfortunate episode,” Sesame Workshop said in a statement on Tuesday.


Clash, 52, the voice of Elmo for nearly three decades, had acknowledged a past relationship with his accuser but said on Monday the pair were both consenting adults at the time. He termed the allegations “false and defamatory.”


“I am a gay man. I have never been ashamed of this or tried to hide it,” Clash said on Monday, saying he was taking a break from the TV show to deal with the situation.


Sesame Workshop said the allegations involving Clash came to its attention in June when the accuser first contacted the company by email. A company executive said it had found “absolutely no evidence that the allegations were true.”


The Elmo character debuted on “Sesame Street” in 1979. While Clash was the third performer to animate the child-like shaggy red monster, Sesame Workshop credits him with turning Elmo into the international sensation he became.


(Reporting By Eric Kelsey and Piya Sinha-Roy, editing by Jill Serjeant and Cynthia Johnston)


TV News Headlines – Yahoo! News



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Recipes for Health: Roasted Eggplant and Chickpeas — Recipes for Health


Andrew Scrivani for The New York Times







Eggplant is always a good, substantial vegetable to use for a vegetarian main dish. The chickpeas and the feta provide plenty of protein. Vegans can leave out the feta and substitute sugar or agave nectar for the honey.




 


1/4 cup extra virgin olive oil


2 garlic cloves, minced


1 28-ounce can chopped tomatoes, with juice, pulsed to a coarse purée


1 teaspoon mild honey (more to taste)


1/4 to 1/2 teaspoon cinnamon, to taste


Salt to taste


1 large or 2 medium eggplants (about 1 1/4 pounds), cut into 1/3-inch-thick slices


3 cups cooked chickpeas (2 cans, drained and rinsed, or, 1 1/2 cups dried – about 3/4 pound


4 ounces feta, crumbled (3/4 cup)


1 teaspoon dried oregano, preferably Greek or Turkish


 


1. Make the tomato sauce. Heat 1 tablespoon of the olive oil in a heavy skillet or wide saucepan over medium heat, and add the garlic. Cook, stirring, until it smells fragrant, about 30 seconds, and add the tomatoes, honey, salt to taste and cinnamon. Cook over medium heat until the tomatoes have cooked down and the sauce is fragrant, about 20 minutes. Taste and adjust seasonings.


2. Meanwhile, heat the oven to 425 degrees. Line a baking sheet with aluminum foil and brush the boil with olive oil. Place the eggplant slices on the baking sheet, salt lightly and brush with olive oil. Place in the oven and bake 20 minutes, or until eggplant is lightly browned and soft all the way through. Remove from the heat. Fold the aluminum foil over and crimp the edges together so that the eggplant steams as it cools. Do this in batches if you need more than one baking sheet. Turn the oven down to 350 degrees.


3. Oil a 2-quart baking dish or gratin. Place the chickpeas in the baking dish and stir in 1 cup of the tomato sauce. Layer the eggplant over the chickpeas and top with the remaining tomato sauce. Sprinkle the feta over the top and drizzle on any remaining olive oil. Sprinkle with the oregano and cover tightly with foil. Bake 30 minutes. Uncover and bake another 10 minutes, until the dish is bubbling.


Yield: 6 servings


Advance preparation: The eggplant slices can be cooked up to a day ahead. Hold in the refrigerator, covered. The tomato sauce will keep for 3 days in the refrigerator and freezes well.


Nutritional information per serving: 366 calories; 16 grams fat; 4 grams saturated fat; 2 grams polyunsaturated fat; 8 grams monounsaturated fat; 17 milligrams cholesterol; 44 grams carbohydrates; 14 grams dietary fiber; 431 milligrams sodium (does not include salt to taste); 15 grams protein


Martha Rose Shulman is the author of “The Very Best of Recipes for Health


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